Signs vs. Symptoms
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Symptom: subjective; what the patient reports (“I have a headache”).
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Sign: objective; what the provider detects (tenderness, rash, splenomegaly, etc.).
Transport Priority
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Wait and treat: minor, manageable cases.
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Load and go: absolute emergencies (e.g., unresponsive w/o gag reflex, cannot follow commands, complicated childbirth, severe bleeding, shock).
Vital Signs Overview
Four core vital signs:
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Pulse (not just heart rate).
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Blood Pressure.
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Respiratory Rate.
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Temperature.
1. Pulse
Definition: Encompasses heart rate, rhythm, volume, character, arterial wall, equality, force, and tension.
Key Arteries:
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Infants: brachial.
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Adults: radial and carotid.
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Avoid palpating both carotids simultaneously → risk of baroreceptor reflex.
Components:
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Rate:
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Normal: 60–90 bpm.
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Tachycardia causes: exertion, fever, anxiety, atropine, epinephrine, anemia, HF, MI, PE, arrhythmias.
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Bradycardia causes: sleep, beta-blockers, calcium channel blockers, vagal stimulation (tight collar, carotid sinus), AV block.
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Rhythm:
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Normal = regular.
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Irregular examples: AFib (irregularly irregular), ectopic beats, digoxin effect.
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Volume (Pulse Pressure):
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Normal pulse pressure ≈ 40 mmHg (120 – 80).
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High: anemia, aortic regurgitation, pregnancy, AV fistula, sepsis, hyperthyroidism.
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Low: low stroke volume or narrowed arteries.
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Character:
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Examples: pulsus alternans, water-hammer (collapsing), bisferiens, paradoxus.
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Equality:
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Compare R vs L arm.
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Unequal = cervical rib, aortic dissection, vascular obstruction.
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Arterial Wall: stiff/calcified in atherosclerosis.
2. Blood Pressure
Definition: Arterial pressure in systemic circulation (not venous/pulmonary).
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Normal: 120/80 mmHg.
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Systolic: pressure during ventricular contraction.
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Diastolic: pressure during ventricular relaxation.
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Mean Arterial Pressure (MAP): average pressure over cardiac cycle.
Formula:
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BP = Cardiac Output × Total Peripheral Resistance (TPR).
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CO = HR × Stroke Volume.
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Stroke Volume depends on contractility + venous return.
Hypertension causes: ↑ HR, ↑ stroke volume, ↑ TPR (vasoconstriction, atherosclerosis).
Hypotension causes: ↓ CO (low HR, low volume), ↓ TPR (sepsis, anaphylaxis).
Isolated Systolic HTN (Wide Pulse Pressure):
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Causes: anemia, AV fistula, pregnancy, sepsis, thyrotoxicosis, Paget’s disease, PDA, acromegaly, beriberi.
BP Cuff Uses:
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Diagnostic (measurement).
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Therapeutic (as a tourniquet in bleeding).
3. Respiratory Rate
Definition: Number of breaths per minute + depth of breaths.
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Normal ventilation = normal rate + normal tidal volume.
Tachypnea causes: exertion, anxiety, aspirin toxicity, anemia, MI, HF, PE.
Bradypnea causes: sleep, opioids, barbiturates, coma, DKA (late).
Ventilation vs. Oxygenation:
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Ventilation = moving air into lungs.
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Oxygenation = transferring O₂ onto hemoglobin.
4. Temperature
Fever (pyrexia):
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Due to infections, pyrogens reset hypothalamic set point.
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Mild = beneficial; prolonged/high = protein denaturation, enzyme dysfunction.
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Drug causes: aspirin, atropine (“aspirin fever,” “atropine fever”).
Hypothermia:
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Dangerous systemic effects:
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Metabolic acidosis (poor perfusion).
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Cell lysis → hyperkalemia, hyperuricemia, hyperphosphatemia.
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Hypocalcemia (due to phosphate rise).
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Insulin resistance → hypoglycemia.
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Coagulopathy (enzymes impaired).
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Splenic sequestration → leukopenia, thrombocytopenia.
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Can precipitate pancreatitis, ↑ hematocrit.
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Causes: exposure, shock, infection, meds (aspirin, atropine).
Clinical Pearls
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Always interpret vitals in context.
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Pulse ≠ just heart rate; it’s a multi-dimensional sign.
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Wide pulse pressure suggests hyperdynamic circulation.
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BP is not static: analyze CO and resistance.
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Resp rate is a sensitive early marker of deterioration.
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Temperature extremes can mask or worsen critical illness.
Summary:
Vital signs, pulse, BP, respiratory rate, and temperature, are foundational in EMS. A structured, nuanced approach (beyond just numbers) helps detect early emergencies, guide management, and prioritize transport decisions.
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