1. Sinus Rhythm
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Normal rhythm: regular P waves followed by QRS complexes.
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PR interval constant.
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QRS = ventricular depolarization, normally narrow (80–100 ms).
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Sinus bradycardia: rate < 60 bpm.
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Sinus tachycardia: rate > 100 bpm.
2. Atrial Fibrillation (AF)
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No distinct P waves, chaotic atrial activity.
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QRS complexes not preceded by P waves.
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Irregularly irregular rhythm (variable R–R intervals).
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Substrate: atrial dilation.
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Trigger: ectopic foci (often pulmonary veins).
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AF with RVR: rate > 100 bpm.
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AF with slow VR: rate < 60 bpm.
3. Atrial Flutter
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Re-entry circuit in atria → coordinated atrial contractions.
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Atrial rate ≈ 300 bpm.
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Narrow complex tachycardia.
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ECG: Sawtooth waves (inverted P waves in inferior leads).
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Conduction ratio: usually 2:1 (ventricular rate ≈ 150 bpm).
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Higher ratios = more AV block.
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1:1 conduction → unstable, risk of VF.
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May mimic AF if conduction ratio varies.
4. Premature Contractions
Premature Ventricular Contractions (PVCs)
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Origin: His–Purkinje system.
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Wide QRS (>120 ms).
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Compensatory pause follows.
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Benign if isolated; risk if frequent (>10–30/hr).
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Patterns:
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Bigeminy (every other beat).
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Runs of PVCs = ventricular tachycardia.
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Premature Atrial Contractions (PACs)
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Abnormal P wave morphology (different from sinus).
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Narrow QRS follows.
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May appear as a pause but timing matches expected P wave.
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Usually benign.
5. Bundle Branch Blocks (BBB)
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General: QRS >120 ms.
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Left BBB:
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Deep S in V1 (“W”).
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Broad R in V6 (“M”).
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Mnemonic: WiLLiaM (W in V1, M in V6).
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Right BBB:
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RSR’ in V1 (“M”).
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Deep S in V6 (“W”).
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Mnemonic: MaRRoW (M in V1, W in V6).
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Note: ST segments altered → difficult ischemia interpretation.
6. AV Blocks
First Degree
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PR interval > 200 ms.
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Usually benign.
Second Degree
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Mobitz I (Wenckebach): Progressive PR prolongation → dropped beat.
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Mobitz II: Dropped beat without PR prolongation.
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Structural issue.
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Risk of progression to complete block.
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Third Degree (Complete Heart Block)
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No association between P waves and QRS.
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Atrial rate > ventricular rate.
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Ventricular escape rhythm → bradycardia.
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Regular P waves + regular QRS, but independent.
7. Ventricular Tachycardia (VT)
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Broad complex tachycardia (>120 ms).
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Monomorphic VT: uniform QRS morphology.
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Polymorphic VT: varying QRS morphologies.
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Torsades de Pointes: polymorphic VT in prolonged QT.
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Features suggestive of VT:
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Very broad complexes (>160 ms).
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Extreme axis deviation.
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No BBB morphology.
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Dangerous: can lead to VF or cardiac arrest.
8. Ventricular Fibrillation (VF)
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No P waves or QRS complexes.
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Chaotic electrical activity, varying amplitudes.
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Minimal cardiac output → lethal without defibrillation.
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Shockable rhythm in cardiac arrest.
9. Cardiac Arrest Rhythms
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Shockable: VT, VF.
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Non-shockable: Pulseless electrical activity (PEA), asystole.
10. ST Elevation
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ST segment: between end of S wave and start of T wave.
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Elevation > 1 mm (except V2–V3 need >1.5 mm).
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Causes:
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STEMI (most important).
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Pericarditis, LBBB, LVH, benign early repolarization.
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Localization:
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Lateral = I, aVL, V5–V6.
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Inferior = II, III, aVF.
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Anterior/septal = V1–V4.
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Posterior = V7–V9.
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Reciprocal changes: ST depression in opposite leads.
High-Yield Mnemonics
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BBB: WiLLiaM (LBBB), MaRRoW (RBBB).
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AF: “Irregularly irregular.”
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Flutter: “Sawtooth.”
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Torsades: Polymorphic VT + long QT.
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