Cardiac Dysrhythmias – Study Notes
General Principles
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EKGs are daunting but manageable with a systematic approach.
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Avoid relying solely on machine interpretations → check with your eyes.
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Use a consistent system (mnemonics like “RAPID PQRST” or Dubin’s method).
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Get comfortable by looking at EKGs often (“pink paper rounds” in the ED).
Basics of EKG
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Small box = 0.04 sec (horizontal) / amplitude = 0.1 mV (vertical).
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Normal sequence: P → QRS → T.
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Intervals: PR, QRS, QT (machine is reliable for these).
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Don’t just read the words on top (the “Ortho interpretation”).
Heart Blocks (AV blocks)
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First-degree AV block
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PR > 200 ms (longer than 5 small boxes).
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Benign; common in young athletes or aging adults.
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No treatment, just note it.
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Second-degree AV block
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Mobitz I (Wenckebach): PR gets progressively longer → dropped beat.
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Often benign; may be medication-related.
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Mobitz II: Constant PR intervals → sudden dropped beat.
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More concerning; risk of progression to complete block.
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Third-degree (complete) block
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P waves march independently of QRS.
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Ventricular escape rhythm slower (~40–60 bpm).
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Always pathologic in acute settings → needs pacing/cardiology consult.
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Premature Atrial Contractions (PACs)
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Extra atrial beat comes early with pause after.
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Usually benign; common after caffeine, alcohol, stress, poor sleep.
Supraventricular Tachycardia (SVT)
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Narrow complex, regular, rapid (often 170–220 bpm).
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Common in young, healthy patients with palpitations.
Management:
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Unstable: Synchronized cardioversion (100 J).
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Stable:
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Vagal maneuvers (Valsalva, REVERT method, carotid massage in kids, ice on face in infants).
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Adenosine 6 mg IV push (warn about transient asystole, chest pressure, dyspnea).
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Calcium channel blockers (verapamil/diltiazem) or beta-blockers if needed.
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Key: If wide and regular → could be VT or SVT with aberrancy. Treat as VT if unsure.
Atrial Flutter
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Atrial rate ~300 bpm; “sawtooth” baseline.
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Ventricular response is divisor of 300 (150 bpm with 2:1 block, 100 with 3:1, etc.).
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Often seen with “holiday heart” (alcohol, bingeing).
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Usually self-limited.
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If unstable: cardioversion.
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If stable: usually no acute treatment.
Atrial Fibrillation (AFib)
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Irregularly irregular rhythm (key finding).
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Two key scenarios:
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New onset: may need workup, cardioversion, anticoagulation.
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Chronic AFib with RVR (>100 bpm): treat like sinus tach → find the trigger (infection, bleed, PE, hypovolemia).
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Don’t reflexively slow rate if tachycardia is compensatory (e.g., bleeding).
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Rate control (beta-blocker, calcium channel blocker, amiodarone) only if indicated.
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If AFib >48h → avoid cardioversion in ED unless unstable (risk of stroke).
Multifocal Atrial Tachycardia (MAT)
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Multiple ectopic atrial foci → ≥3 different P-wave morphologies.
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Irregular, narrow tachycardia.
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Classically seen in COPD patients.
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Treat underlying condition (O₂, bronchodilators), not the rhythm itself.
Wolff-Parkinson-White (WPW) Syndrome
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Accessory conduction pathway → risk of rapid ventricular rates.
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EKG clues:
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Short PR interval.
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Delta wave (slurred upstroke).
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Wide QRS.
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Management:
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Narrow, regular SVT → treat like typical SVT.
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Wide, irregular AFib with WPW → very dangerous.
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Avoid AV nodal blockers (adenosine, diltiazem, beta-blockers, digoxin).
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Use procainamide (safe for all wide-complex tachycardias if unsure).
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Wide Complex Tachycardia (WCT)
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Assume ventricular tachycardia (VT) until proven otherwise.
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Unstable: cardioversion.
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Stable:
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If uncertain → treat as VT.
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Procainamide is drug of choice for stable WCT.
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Key Clinical Pearls
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Always assess stability first.
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If unstable → shock (synchronized if organized rhythm).
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For wide tachycardia: safer to assume VT.
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Adenosine is diagnostic and therapeutic for narrow SVT but never give in AFib with WPW.
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Chronic AFib with RVR is often physiologic tachycardia—look for the cause before treating the rate.
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