Friday, September 26, 2025

Cardiac Dysrhythmias – Study Notes

General Principles

  • EKGs are daunting but manageable with a systematic approach.

  • Avoid relying solely on machine interpretations → check with your eyes.

  • Use a consistent system (mnemonics like “RAPID PQRST” or Dubin’s method).

  • Get comfortable by looking at EKGs often (“pink paper rounds” in the ED).

Basics of EKG

  • Small box = 0.04 sec (horizontal) / amplitude = 0.1 mV (vertical).

  • Normal sequence: P → QRS → T.

  • Intervals: PR, QRS, QT (machine is reliable for these).

  • Don’t just read the words on top (the “Ortho interpretation”).

Heart Blocks (AV blocks)

  1. First-degree AV block

    • PR > 200 ms (longer than 5 small boxes).

    • Benign; common in young athletes or aging adults.

    • No treatment, just note it.

  2. Second-degree AV block

    • Mobitz I (Wenckebach): PR gets progressively longer → dropped beat.

      • Often benign; may be medication-related.

    • Mobitz II: Constant PR intervals → sudden dropped beat.

      • More concerning; risk of progression to complete block.

  3. Third-degree (complete) block

    • P waves march independently of QRS.

    • Ventricular escape rhythm slower (~40–60 bpm).

    • Always pathologic in acute settings → needs pacing/cardiology consult.

Premature Atrial Contractions (PACs)

  • Extra atrial beat comes early with pause after.

  • Usually benign; common after caffeine, alcohol, stress, poor sleep.

Supraventricular Tachycardia (SVT)

  • Narrow complex, regular, rapid (often 170–220 bpm).

  • Common in young, healthy patients with palpitations.

Management:

  • Unstable: Synchronized cardioversion (100 J).

  • Stable:

    • Vagal maneuvers (Valsalva, REVERT method, carotid massage in kids, ice on face in infants).

    • Adenosine 6 mg IV push (warn about transient asystole, chest pressure, dyspnea).

    • Calcium channel blockers (verapamil/diltiazem) or beta-blockers if needed.

Key: If wide and regular → could be VT or SVT with aberrancy. Treat as VT if unsure.

Atrial Flutter

  • Atrial rate ~300 bpm; “sawtooth” baseline.

  • Ventricular response is divisor of 300 (150 bpm with 2:1 block, 100 with 3:1, etc.).

  • Often seen with “holiday heart” (alcohol, bingeing).

  • Usually self-limited.

  • If unstable: cardioversion.

  • If stable: usually no acute treatment.

Atrial Fibrillation (AFib)

  • Irregularly irregular rhythm (key finding).

  • Two key scenarios:

    • New onset: may need workup, cardioversion, anticoagulation.

    • Chronic AFib with RVR (>100 bpm): treat like sinus tach → find the trigger (infection, bleed, PE, hypovolemia).

  • Don’t reflexively slow rate if tachycardia is compensatory (e.g., bleeding).

  • Rate control (beta-blocker, calcium channel blocker, amiodarone) only if indicated.

  • If AFib >48h → avoid cardioversion in ED unless unstable (risk of stroke).

Multifocal Atrial Tachycardia (MAT)

  • Multiple ectopic atrial foci → ≥3 different P-wave morphologies.

  • Irregular, narrow tachycardia.

  • Classically seen in COPD patients.

  • Treat underlying condition (O₂, bronchodilators), not the rhythm itself.

Wolff-Parkinson-White (WPW) Syndrome

  • Accessory conduction pathway → risk of rapid ventricular rates.

  • EKG clues:

    • Short PR interval.

    • Delta wave (slurred upstroke).

    • Wide QRS.

Management:

  • Narrow, regular SVT → treat like typical SVT.

  • Wide, irregular AFib with WPW → very dangerous.

    • Avoid AV nodal blockers (adenosine, diltiazem, beta-blockers, digoxin).

    • Use procainamide (safe for all wide-complex tachycardias if unsure).

Wide Complex Tachycardia (WCT)

  • Assume ventricular tachycardia (VT) until proven otherwise.

  • Unstable: cardioversion.

  • Stable:

    • If uncertain → treat as VT.

    • Procainamide is drug of choice for stable WCT.

Key Clinical Pearls

  • Always assess stability first.

  • If unstable → shock (synchronized if organized rhythm).

  • For wide tachycardia: safer to assume VT.

  • Adenosine is diagnostic and therapeutic for narrow SVT but never give in AFib with WPW.

  • Chronic AFib with RVR is often physiologic tachycardia—look for the cause before treating the rate.

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